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Download e-book for kindle: Dyslipidemias: Pathophysiology, Evaluation and Management by Abhimanyu Garg

By Abhimanyu Garg

ISBN-10: 1607614235

ISBN-13: 9781607614234

ISBN-10: 1607614243

ISBN-13: 9781607614241

Dyslipidemias: Pathophysiology, review and Management presents a wealth of normal and particular guidance for the scientific overview and administration of lipid issues in adults and youngsters. overlaying the whole variety of universal via infrequent lipid issues, this well timed source bargains specified, useful details for all clinicians who deal with sufferers with dyslipidemias, together with normal internists, pediatric and grownup endocrinologists, pediatricians, lipidologists, cardiologists, internists, and geneticists. For the final 20 years, there was a transforming into acceptance around the globe of the significance of handling dyslipidemia for the first and secondary prevention of atherosclerotic vascular illness, specifically heart illness. This has been typically as a result book of the information of nationwide ldl cholesterol schooling Program’s grownup therapy Panel and Pediatric Panel from the U.S.. those guidance have motivated iteration of comparable options from worldwide, rather Europe, Canada, Australia and Asia. constructed by means of a popular crew of top overseas specialists, the booklet deals cutting-edge chapters which are peer-reviewed and characterize a entire evaluation of the sphere. a tremendous addition to the literature, Dyslipidemias: Pathophysiology, review and Management is a gold-standard point reference for all clinicians who're challenged to supply the easiest care and new possibilities for sufferers with dyslipidemias.

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Additional info for Dyslipidemias: Pathophysiology, Evaluation and Management

Example text

Automated protein immunoassays were developed and apolipoprotein B was shown to be highly associated with LDL-C and greater CVD risk, especially in European studies [51, 52]. Concentrations of apolipoprotein A1 were highly associated with HDL-C and higher levels of each appeared to be cardioprotective. In analyses that compared prediction models with LDL-C and HDL-C versus models with apolipoprotein B and apolipoprotein A1, the overall ability to discriminate was similar. The results were interpreted as showing that measurement of apolipoproteins did not improve estimation beyond the traditional analytic approach with total cholesterol and HDL-C to estimate risk for initial CVD events [38].

33. Durrington P. Dyslipidaemia. Lancet. 2003;362 (9385):717–31. 34. Brunzell JD, Schrott HG, Motulsky AG, et al. Myocardial infarction in the familial forms of hypertriglyceridemia. Metabolism. 1976;25:313–20. 35. Siegel RD, Cupples A, Schaefer EJ, Wilson PW. Lipoproteins, apolipoproteins, and low-density lipoprotein size among diabetics in the Framingham offspring study. Metabolism. 1996;45(10):1267–72. 36. Garrison RJ, Kannel WB, Feinleib M, Castelli WP, McNamara PM, Padgett SJ. Cigarette smoking and HDL cholesterol: the Framingham offspring study.

7, principal components analysis showed that the metabolic syndrome traits clustered. The presence of three or more of the traits typically led to a doubling or tripling of risk for CVD, and more than a 20fold greater risk for diabetes mellitus [42, 43]. A variety of other plasma biomarkers were subsequently used to study these phenomena, including laboratory biomarkers, traditional lipoprotein cholesterol levels, smaller LDL particles, and greater LDL particle number [28, 44–48]. Apolipoproteins Lipoprotein particles include apolipoproteins, cholesterol, triglycerides, and phospholipid moieties.

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Dyslipidemias: Pathophysiology, Evaluation and Management by Abhimanyu Garg

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