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Clinical Endocrine Oncology by Ian D. Hay, John A. H. Wass PDF

By Ian D. Hay, John A. H. Wass

ISBN-10: 1405145846

ISBN-13: 9781405145848

A really accomplished reference for the administration of sufferers with endocrine melanoma

The re-creation of medical Endocrine Oncology has been totally revised and prolonged making it the main finished and updated reference to be had. Written and edited through prime overseas specialists within the box, it units the normal in multidisciplinary deal with sufferers with endocrine tumors.

The ebook presents particular and distinct tips at the simple, scientific, investigative and healing techniques required for the thorough overview of a sufferer with a tumor in an endocrine organ. The eighty-four chapters are prepared in seven components:

• Endocrine Oncology and healing innovations • Thyroid and Parathyroid Tumors • Pituitary and Hypothalamic Lesions • Adrenal and Gonadal Tumors • Neuroendocrine Tumors and the medical Syndromes • scientific Syndromes and Endocrine Neoplasia • Endocrine-responsive Tumors and feminine Reproductive Hormone treatment.

This authoritative and useful textual content should be a useful source for all these operating within the box, together with endocrinologists, scientific oncologists, surgeons, radiation therapists, interventional radiologists, expert nurses, and medical scientists.

John A.H. Wass is joined during this variation by means of a brand new editor, Ian D. Hay, Professor of medication and Endocrinology study on the Mayo health center collage of medication, Rochester, Minnesota, united states.

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Extra info for Clinical Endocrine Oncology

Sample text

However, occipital neural crest cells migrate through the 3rd and 4th arches and all three tissues express the ret (receptor-type tyrosine kinase) proto-oncogene. Parathyroid hormone-related protein This molecule, which acts like PTH on the PTH receptor, is produced by a wide variety of tumors (particularly lung and breast) and is the cause of the hypercalcemia of malignancy. It is also secreted constitutively by a similarly wide variety of fetal and adult tissues. Its physiological autocrine and paracrine function in these tissues has yet to be determined.

4). They are usually said to arise from the pouch endoderm but more recently it has been suggested that they arise from the adjacent cleft ectoderm. Parathyroid 3 arises as a cellular mass from the dorsal and the thymus from the ventral part of the 3rd pharyngeal pouch. As the thymus descends into the anterior mediastinum parathyroid 3 is carried part-way with it so that it forms the inferior parathyroid gland. It is much more variable in position than parathyroid 4 which becomes the superior parathyroid.

Its floor comprises the optic chiasm and tract, the pituitary stalk, mamillary bodies and posterior perforated substance; anteriorly it is limited by the lamina terminalis and anterior commissure; posteriorly it blends with the midbrain; superiorly it is continuous with the thalamus and epithalamus; and laterally it blends with the zona incerta and internal capsule. Its neuroendocrine neurons are situated in its medial zone (Fig. 2): magnocellular neurons secreting vasopressin or oxytocin as their primary product are located mostly in the supraoptic and paraventricular nuclei; parvocellular neurons secreting the releasing factors that control the anterior pituitary are more widely distributed but many are located in the ventromedial part of the paraventricular nucleus (CRH, thyrotropin-releasing hormone (TRH)), in the arcuate nucleus (dopamine, growth hormonereleasing hormone (GHRH)), in the adjacent medial basal hypothalamus (gonadotropin-releasing hormone (GnRH)) and in the periventricular zone (somatostatin).

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Clinical Endocrine Oncology by Ian D. Hay, John A. H. Wass

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